MRI cardiac morphology function and velocity flow w/wo contrast..

Posted by lpaelevated4 @lpaelevated4, Oct 16 5:54am

I had 2 echocardiograms and first showed mild left ventricular posterior wall thickness of 1.1 cm and septal wall thickness of 1.2 cm. I was asked for a sleep apnea lab test(negative result) and to quit exercising for 3 months to see if mild lvh thickness would decrease. It did and both Left ventricular posterior and septal wall thickness were 1.0 cm , but still classified as mild LVH.
Saw Cardio a couple of days ago and he ordered a MRI cardiac for me. Is this the most accurate measurement for Left ventricular posterior and septal thickness walls?
Will the MRI report comment on my aorta as the first echo showed no stenosis and regurgitation but the 2nd showed no significant stenosis. The first report showed no mitral and triscupid regurgitation but the 2nd report showed trace of regurgitation.
Also I have high iron via blood test but MRI liver showed normal iron level. Could the extra iron be deposited in my heart and harm me? Will Cardiac MRI detect this?

TIA

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gloaming | @gloaming | Oct 16 4:00pm

I don't know if this will be helpful to you:
https://ashpublications.org/blood/article/116/21/SCI-27/69908/Multiple-Causes-of-Iron-Overload-In-Tissues-Cells
It turns out that hemochromatosis can cause problems for the organs, especially the kidneys and heart. I would expect it to be close to linear at first, but there would come a point where it gets more problematic much more quickly. That's just a guess. But for mild hyper uptake of iron, say from the diet, it should only have an effect after a considerable time, perhaps months.
As you know, exercise can cause some enlargement of the heart's chambers, but so can hypertension and arrhythmias, especially atrial fibrillation (AF). If those are negative, then it's idiopathic and the physicians may or may not be able to do much except to try medications....which you may have to take for the rest of your life.
As we age, our hearts change, and almost without exception it's never for the better. We get more pathology and the valves become stenosed or prolapsed and those bring a decrease in the aortic flow and increased regurgitation into the sister chamber.

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