Eliquis and AFIB

@chanemann1 My new EEP told me that they are moving toward a consensus that blood thinners will become an 'as needed' therapy for those who fit criteria. I now carry it with me as I also had only one episode over 19 months ago. He said a 'situational' afib or aflutter occurance is common after a viral disease, covid and severe respiratory event. It does not mean you have either, it means you had them once. He said medicine as a whole moves slowly, the REACT trial should move it closer to this conclusion. In the meantime I am happy to carry and not take the pills.

@emo44 Thank you very much for, I appreciate the response! Most likely I am going to go on the Eliquis, but we must remember Eliquis is not 100% that we would not form a cloth and no one can claim 100%. I think it’s 70% of course better than no protection at all thank you good luck to you also.

Is there anything bad with taking Eliquis? I have been on it for over 16 months, only issue Bruzing

I don’t know why people that have Afib wouldn’t want to take Eliquis or a blood thinner. Having Afib and not taking a blood thinner is like playing roulette with your life. For anyone that has seen the devastating effects of a massive stroke, taking Eliquis is a blessing. It is the least of my worries with Afib and actually soothing to know I won’t most likely have a stroke. Multaq on the other hand was a nightmare for me and sent me to the hospital in congestive heart failure. It has left me in almost constant Afib after having to stop taking it. I wouldn’t take any of the other meds they have except amiodarone for conversion. I wouldn’t take it constantly.

@chickenfarmer I would take Eliquis as long as I have any Afib. If you had ever seen anyone that had a massive stroke from not using a blood thinner, you would know why. Good luck.

THE trouble with all prescriptions is that we are all different. The GENERAL TREND of Doctors at the moment is to completely ignore and deny the side effects of Eliquis ( other drugs as well) . Denying and Ignoring these side effects are harmful to the people who have had to "document " their HEALTH ISSUE .
That is the problem. How do those people recover from the harm this or other drugs have created for them.

THERE is no doctor for the people who know that they must recover from a drug that was given to them .

Yes we are all Different.
Doctors All across the Board are OVER WHELM by today’s DEMANDS OF BIG CORPs.
they went to School Studying for Years & long HOURS to be able to HELP us with ALL Our Health Issue thats how we have made Progress.

Time for Clot Formation in the Left Atrial Appendage
Clot formation in the left atrial appendage (LAA) during atrial fibrillation (AF) can begin almost immediately, but a clinically significant thrombus is most often considered likely after about 48 hours of continuous AF — though the process is gradual and varies by individual risk factors

How the process works
In normal heart rhythm, the LAA contracts efficiently and blood flow is relatively brisk. In AF, the atrium quivers instead of contracting, and blood in the LAA slows to near stasis. This pooling activates clotting proteins and allows a thrombus to form.

The LAA is the most common site because of its small size, complex internal folds, and low flow velocity even under normal conditions.

Time course
Immediate risk: Blood stasis in the LAA can start right after AF begins, but clot growth is slow and depends on flow conditions

48‑hour clinical rule: Guidelines have long used 48 hours as a threshold — AF lasting less than two days is considered lower risk for a mature clot, while >48 hours warrants anticoagulation or imaging before restoring normal rhythm .

Short episodes: A 10‑minute AF episode is unlikely to produce a clinically dangerous clot

Persistent AF: In permanent AF, clot risk is higher and can develop over days to weeks.

Influencing factors
AF duration and frequency — longer and more frequent episodes increase risk.

Patient risk profile — age, hypertension, diabetes, prior stroke, heart failure, and high CHA₂DS₂‑VASc score raise clot risk

Flow dynamics — chamber dilation and reduced blood refreshment in the LAA during AF slow clot formation.

Anticoagulation — therapeutic anticoagulants can prevent or delay clot formation.

Clinical implications
Because clot formation is not instantaneous, short AF episodes may not produce a clot, but after 48 hours the likelihood rises significantly, which is why cardioversion is generally safe without anticoagulation only within that window.

.For AF of unknown duration or >48 hours, guidelines recommend at least 3 weeks of anticoagulation before cardioversion.

In summary: Clotting in the LAA can start almost immediately in AF, but a clinically significant thrombus is most often considered likely after ~48 hours of continuous AF, with risk increasing over days to weeks depending on patient factors and AF persistence

I wear a Fitbit, and at night it shows AFib some times for 30 minutes