I'm not going to keep checking it. It's so high, and calcification isn't generally something that goes away in any tissue, I'm just resigned to it. But I'm guessing that it's not intimal, ie, it does NOT represent atheroma load. I, like you, have normal treadmill and perfusion studies. My horrendous allergy to contrast dye makes an elective angio out of the question. But we could have 60% lesions with no substantial flow deficit, so we'd seem fine WITH coronary artery disease. If that's the case, our best shot at dodging the reaper is to prevent further atheroma formation with exactly what we're doing. I'm not sure more exercise will prevent anything as I've been an exerciser - runner/strength, etc - all my adult life, and here we are. In my case, and you can't really use this for any sort of comparison, I have extremely high antibody levels to two species of Bartonella, a nasty bug that attacks endothelium (part of my Lyme plague). Inflammation there might very well be the stimulus for calcification. That whole facet of my medical story is not for here.... but, I'm working on it. Onward~~~~~~~>>. jon

btw, pcs - statins rarely do liver damage and when detected by regular lab monitoring, can often be mitigated by dose or type of statin change. In my case, I DID get liver and muscle injury from lipitor but NOT from Livalo. Details...... As always, choose your genes wisely and try not to age...... 🥴