There is a difference between triggers of vagotonic and adrenergic Afib events. They also may respond differently to ablations. Literature studies in NIH data base PUB MED https://pubmed.ncbi.nlm.nih.gov and Google Scholar may help you find studies that show the efficacy of ablation and or drugs on adrenergic Afib vs Vagotonic. Adrenergic Afib is particularly set off by exercise and higher heart rates and is a result of fibrous.
See "Elevated β1-Adrenergic Receptor Autoantibody Levels Increase Atrial Fibrillation Susceptibility by Promoting Atrial Fibrosis"
https://shorturl.at/x65K2
Vagotonic is set off by stimulation of the vagus nerve often positional. A good EP cardiologist should be able to explain the differences of the 2 and what if any outcome differences there might be to an ablation. Drug therapy, often Sotalol is used to keep the HR lower thus reducing Afib triggers. The down side is that it is harder to get in a good work out with Sotalol because it suppresses HR. Also you have to monitor yourself when exercising. An example of this is my brother who at 81 walks 4 miles/day and lift weights but sometimes he pushes too hard and it will trigger Afib. He has used it successful for about 4 years but recently has had a couple of longer Afib events suggesting that the drug is becoming less helpful.
I myself very recently underwent a 2nd ablation after my first ablation 5 1/2 years ago was showing signs of increased Afib events usually lasting < 36 hours. In the first 4 years after the 1st ablation I had 3 episodes. Then last fall I started having them on average monthly.