Auto accident cervical - pinched nerve and spinal cord herniation

I am looking for a Neurosurgeon. I was in a vehicle accident 8/2024. MRIs show herniations at C3-C4, C4-C5, C6-C7, Nerve Root Compression (Radiculopathy), and Spinal Cord Compression (Myelopathy), abnormal neck curvature (kyphosis) and vertebral slip (retrolisthesis), foraminal stenosis (narrowing of the nerve exits) at C3-C4, C4-C5, and most significantly C5-C6, and 3 small lumbar herniations. I had X-ray guided steroid injections in my cervical and lumbar. Lumbar didn't help, so we did a nerve block. 10/2024 I had my left carpal and left thumb ligament reattachment surgeries. 4/2025 I had right carpal surgery. I was on pregabalin and meloxicam 12 months and also steroids by my Pain management doctor. I don't like taking pain medication so I take intermittently. I have tingling in my fingers, trouble carrying anything over 7 lbs, drop things, my hot/cold sensations don't work properly, my balance is slightly off. I'm looking for someone to explain my MRI in lame-mans terms. I'm 45 trying to decide which route to go picking a Doctor.

PROCEDURE: MRI CERVICAL SPINE WITHOUT CONTRAST
TECHNIQUE: Multisequence, multiplanar MRI obtained on a high-field 1.5 T MRI
system
CLINICAL INDICATION: Neck and back pain, thumb/wrist pain; history of motor
vehicle collision 8/2/2024
COMPARISON: No prior studies available for comparison
FINDINGS:
There is abnormal reversal of the lordotic curvature which may be seen in
conjunction with posttraumatic myospasm, articular dysfunction or other
biomechanical abnormality. In addition, it has been suggested that cervical
kyphosis may be associated with abnormal increased load on the spinal cord
and/or nerve roots. Advise clinical and radiographic correlation.
C2-C3: Disc height is adequate. Posterior elements are normal. No evidence of
focal herniation. No significant stenosis or neural mass effect observed at
this time.
C3-C4: Dorsal, left asymmetric 1-1.5 mm herniation. Adequate disc height.
Facet joints are normal. No significant stenosis or spinal cord or nerve root
compromise.
C4-C5: Right paramedian herniation measuring approximately 2 mm. Normal
posterior elements. Disc height is adequate. No foraminal stenosis exiting
nerve root compromise. There is encroachment of the right anterolateral spinal
cord margin without spinal cord compression.
C5-C6: Disc height narrowing with 3-4 mm broad-based uncinate
proliferative-herniation complex observed. Grade 1 retrolisthesis of C5. There
is posterior element hypertrophy. Combined changes creating ventral spinal
cord abutment and low-grade compression asymmetric to the left. Neural
foraminal stenosis observed. Clinical correlation for posttraumatic C6
radicular involvement or myelopathy recommended.

C6-C7: Disc height is adequate. Posterior elements are normal. No evidence of
focal herniation. No significant stenosis or neural mass effect observed at
this time.
C7-T1: Disc height is adequate. Posterior elements are normal. No evidence of
focal herniation. No significant stenosis or neural mass effect observed at
this time.
No compression fracture. Marrow signal alteration demonstrating hyperintensity
on fluid sensitive images and low signal on T1 seen at the opposing C5-C6
vertebral margins thought to represent Modic type I reactive inflammatory
changes; however, given the patient's history of recent injury posttraumatic
contusion could be considered as well. No aggressive vertebral or marrow
lesion identified.
There is no evidence of intramedullary or intradural masses or signal
alteration. The spinal cord is of normal caliber. Atlantoaxial level is normal
in appearance and the craniocervical junction is unremarkable without evidence
of cerebellar tonsil herniation. No specific abnormality of the paraspinal
musculature. Normal vascular flow voids are noted. The remaining
paravertebral soft tissues are normal in appearance to the extent visualized.
CONCLUSIONS:
1. Segmental changes involving the discovertebral junctions and/or posterior
elements as detailed in the level-by-level analysis above.
2. C5-C6 neural encroachment-compromise as discussed. Please see above for
more detailed segment specific descriptions. Recommend clinical correlation
for associated posttraumatic radicular involvement and/or myelopathy as above.
3. Abnormal cervical kyphosis as discussed in the opening paragraph above.
Clinical and radiographic correlation advised for posttraumatic myospasm,
articular dysfunction and/or other abnormal spinal biomechanics.
4. The retrolistheses at C5-C6 suggest possible underlying injury and/or an
impairment of the anterior longitudinal ligament at this level. No acute edema
or full-thickness ligament tear is visible on this exam.
5. C5-C6 type I Modic changes versus posttraumatic contusion as discussed
above.