@osteocurious
Well, the time for the fracture to heal is concerning, but it healed and that is what is most important.
That dexa is not bad compared to many of us. Yes TBS will give you some good information and peace of mind too, I hope.

Now even more interesting, I am seeing where strontium citrate is not bound by the 25-hydroxylation in the liver to become 25(OH)D, and then activation in the kidneys. In other words, this earth mineral, strontium, that acts like calcium in our body bypasses the problem which your use of antiseizure drug is causing by blocking or slowing your calcium uptake.

Strontium’s anabolic effect is not strictly dependent on the Vitamin D pathway.
Strontium accelerates the filling of bone defects and improves cortical/trabecular microarchitecture.
Long-term (up to 10 years) strontium use has shown sustained increases in BMD at the femoral neck.

To say it another way: Strontium functions differently than calcium because it primarily exerts its effects by acting directly on bone cells. While calcium's metabolism is heavily dependent on vitamin D levels—which are frequently depleted by the liver enzymes induced by certain antiseizure medications (ASMs)—strontium does not appear to rely on those same pathways.

I am a strontium citrate user and am now in normal bone density and normal bone quality.
I have research on strontium if you are interested.