Connect
← Return to treatment options for paratracheal lymph nodes with BRCA2
Discussion
treatment options for paratracheal lymph nodes with BRCA2
Prostate Cancer | Last Active: 2 days ago | Replies (19)Comment receiving replies
@edmond1971
The problem with depending on the drugs to handle actual Metastasis is not a long-term solution. The drugs will not last for the long-term and your PSA will start to rise, even though you are on them, and the metastasis will start to grow. Your SUVs were very low so you may not want to even do anything at all about them.
I can’t imagine getting off ADT and having my PSA rise to 19. That just is not careful planning. I know people who have BRCA2 and have gone on vacations and other people who do not have it but are Gleeson, nine who have gone on vacations, and in all these cases, their metastasis have increased dramatically after a second or third vacation. Sounds like a great idea, but it has not worked out well for people I know. I attend nine advanced prostate cancer online meetings every month so I hear from a lot of people.
As for the PARP inhibitor. My oncologist has discouraged my getting on a PARP inhibitor Until the other ADT and ARPI drugs stop working. There’s good reasoning behind this. I didn’t disagree with her because I was paid to review Akeega documentation. That drug includes a PARP inhibitor with Zytiga. The warnings In the documentation, Which my oncologist referred to, We’re pretty eye-opening. The PARP inhibitor causes serious problems with your blood. RBC, WBC and platelets tests can be dramatically reduced resulting in people needing blood transfusions because they are so anemic. It also can cause high blood pressure.
These are things to discuss with your doctors before going on a PARP. I’m staying on ADT and Nubeqa Until it stops working. Then I will take a PARP.
Replies to "@edmond1971 The problem with depending on the drugs to handle actual Metastasis is not a long-term..."
Connect
@jeffmarc
\\ I attend nine advanced prostate cancer online meetings every month so I hear from a lot of people
Jeff, why do you think, in the same situations (age, Gleason score, stage) and with roughly the same treatment, metastases grow in some people, while in others they simply remain dormant? What influencing factors would you identify? For example: place of residence, physical activity, diet, obesity, immune system characteristics, certain body factors (for example, prolactinin and insulin compete with testosterone), anything that interferes with testosterone synthesis, and other factors. Which of these are significant for creating a map of recommendations on what should be done and what, conversely, should not be done?
This information needs to be somehow classified, processed, and analyzed, and I think that if conclusions were drawn from this analysis, it would be valuable for those who are sick. There are no universal methods, but there are general patterns.
In my life, I've met people who had recovered from late-stage cancer, and when I asked (I've been very curious since childhood) what they did, they said that first of all, they stopped being nervous, let go of resentments, and started moving frequently, because blood is the river of life, and it is it that brings possible salvation.
I lived life to the fullest, but now I'm so crushed and sad that I've become very withdrawn and gloomy. This suddenly befell me due to a doctor's mistake, who missed important tests. I used to be obsessed with revenge, but lately I've taken the blame. It's hard to understand when nothing indicates a problem and then suddenly it appears with frequent trips to the bathroom.