leslie, you are so right.
The primary mechanism action with Evenity is anti resorptive. You probably already know that it targets sclerostin and prevent the cells that break bone down from developing. It blocks the normal process of bone remodeling. It does also add bone, the way that Prolia adds bone. While they are both classified as anti resorptive because of their (different) primary mechanisms, they occupy a space somewhat in between the terms anabolic and anti resorptive.
It's bone that is added scaffold-like to the outer portion of existing bone, covering fissured bone without resolving fissures or restoring blood supply.
While I wouldn't call either Prolia or Evenity anabolic, there isn't a reason to quibble over terminology. Further, since the purpose of all of these pharmaceuticals is to prevent fracture, it might be enough that they all do so.
There aren't good studies about how fast the bone added by Evenity is lost after stopping the medications. And I hope there won't be case reports because we should all be guided into a follow up medication.
The bone added is also heavy bone unarticulated by cannulation needed to change the bone internally according to our usual activities. This bone can't be as resilient as anabolic or parathyroid-bone.
I appreciate your post