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Post-COVID Recovery & COVID-19 | Last Active: Aug 18 6:48pm | Replies (34)
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Just a query from fellow 70 year old hypothyroid sufferer still with LC…did LC affect your hypothyroidism, as you suggest it did but what do you mean by ‘meds increased my TSH from 3 to 1’…surely this is a decrease, changed by hormone replacement or are you using other ‘meds’? All specialists drs.have washed their hands of me unfortunately and I am left with ‘agreeing to disagree’ with my GP who thinks it’s ’overmedicated on levothyroxine’ that is giving me my high HR…but I do take a combo T4/T3 and have always had a suppressed TSH (0.06-0.02). It’s the postural hypotension on standing and hypertension at rest that is new( well 4 1/2 years) to LC that is disabilitating.
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LC did not influence my hypothyroidism. Thyroid stimulating hormone (TSH) normal ranges from about 0.4 and 4.5. A number "less than" 0.4 is high levels of TSH (a bit hard to wrap one's brain around this) and can be borderline hyperthyroid. Increasing one's Synthroid or other thyroid meds results in lower TSH numbers, which are actually higher levels. I have had to do the WWW smackdown with doctors that think a TSH level of 3 is fine; I tell them that a (higher) level of 1 to 2 gives me more energy, and better (GI) regularity. So, I finally increased my levothyroxine from 75 mcg/day, to that plus one 88 mcg once a week, which moved me from 3 to 0.9 TSH. A TSH of 0.06 to 0.02 sounds close to hyperthyroid range, which can be associated with cardiovascular symptoms. At least for me, a TSH level of 0.15 resulted in irregular heat beats.