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@gloaming

The statins are like DOACs...Direct-acting Oral Anti-Coagulants. The DOACs don't prevent clotting, they merely retard the action so that your heart can flush out 'stale' blood before it actually clots if it is fibrillating and the left atrial appendage is not getting 'irrigated' quite as well as when one's heart is in normal sinus rhythm. Statins are somewhat similar, but they also have some unfortunate 'side-benefits', such as preventing your body from making its own co-enzyme Q10. If you simply must take them, statins reduce the liver's ability to produce low-density lipids, but they don't absolutely stop all such production. So, depending on the dosage (and now you realize that the answer is dependent on one's choice of food as well as her willingness to take the prescribed dosage of statin), you might stop 80% or only 40% of your liver's production of LDL.
https://www.healthcentral.com/article/can-you-eat-whatever-you-want-while-taking-statins-for-cholesterol

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Replies to "The statins are like DOACs...Direct-acting Oral Anti-Coagulants. The DOACs don't prevent clotting, they merely retard the..."

I'm afraid I don't understand your reply, but at least you tried to address my question, as opposed to the first response. The article you posted also doesn't answer my question (I actually read it earlier today in hopes of answers while googling).

Let me rephrase in a diffent way: If two patients have the exact same cholesterol numbers, BP control, healthy weight, active lifestyle, and all the rest is identical, is the one who eats well any less likely to suffer a cardiac event than the one who eats poorly? And if yes, how do we know this and how might we measure it? Would a doctor be able to tell which is which, and if so, how would they know?