Prednisone 10 mg/day for about 12 years following serendipitous discovery: Out of concern that new, severe tinnitus might be a harbinger of immune-mediated acoustic neuropathy with a risk of hearing loss, I took high-dose prednisone for about a week. I don't recall any immediate response of tinnitus, but to my surprise, prednisone promptly abolished my peripheral neuropathic pain. Subsequently, the pain recurred whenever, on multiple occasions, I tapered prednisone below 10 mg/day. As time has gone by, I have continued prednisone 10 mg/day plus, with increasing severity and distribution of pain, I have tried multiple pharmacological interventions (plus a spinal cord stimulator) to control pain and/or neuropathic processes. Only recently have I (with physician concurrence) hit upon trying higher-dose prednisone burst as described earlier.

My continued use of prednisone found academic support in the following report by Dabby:

Acute steroid responsive small-fiber sensory neuropathy: a new entity?
Ron Dabby, Ronit Gilad, Menachem Sadeh, Yair Lampl, Nathan Watemberg. First published: 07 March 2006 https://doi.org/10.1111/j.1085-9489.2006.00062.xCitations: 46.
"Abstract: Small-fiber neuropathy is often idiopathic and commonly follows a chronic course. Treatment is often effective in treating the core symptom of pain, but it has no effect on the pathologic process. We describe four patients with acute small-fiber neuropathy who responded dramatically to steroid therapy. All patients had acute onset neuropathic pain, normal nerve conduction studies, and evidence of small-fiber dysfunction in quantitative sensory testing and skin biopsy. Symptoms were distal and symmetrical in three patients and generalized in one patient. In two cases, the neuropathy presented as an erythromelalgia-like syndrome. Marked clinical improvement occurred 1–2 weeks after oral prednisone therapy was initiated. Three patients remained symptom free, and one patient experienced recurrence of neuropathy after prednisone was tapered."